1. Immune factors accounted for about 20 percent in the blood of such antibodies can be detected in patients with ovarian often combined in other autoimmune diseases such as lupus erythematosus rheumatoid arthritis congenital lack of an enzyme in the body
2. Cytogenetic factors reproductive cells less congenital atresia speed up the follicle X chromosome abnormalities in patients with a family history of 10%
3. Physical factors such as surgery chemotherapy infection caused gonadotropin secretion defect, or gonadotropin receptors or receptor defects
4. Idiopathic premature ovarian failure patients with no clear reasons for the normal chromosomes of blood sex hormone FSH (follicle stimulating hormone) LH (luteinizing hormone) increased E2 (estrogen) lower PRL (PRL) in patients with normal estrogen not only have the potential lack of Ovarian androgen production or lack of adrenal steroid hormones ability to generate mild defects laparoscopy small ovarian or ovarian atrophy
2. Cytogenetic factors reproductive cells less congenital atresia speed up the follicle X chromosome abnormalities in patients with a family history of 10%
3. Physical factors such as surgery chemotherapy infection caused gonadotropin secretion defect, or gonadotropin receptors or receptor defects
4. Idiopathic premature ovarian failure patients with no clear reasons for the normal chromosomes of blood sex hormone FSH (follicle stimulating hormone) LH (luteinizing hormone) increased E2 (estrogen) lower PRL (PRL) in patients with normal estrogen not only have the potential lack of Ovarian androgen production or lack of adrenal steroid hormones ability to generate mild defects laparoscopy small ovarian or ovarian atrophy
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