Jul 28, 2008

Hypertrophy of the uterus

[Outlined]

Hypertrophy of the uterus (hypertrophy of uterus), the uterus is even greater, muscle thickness of 2.5 cm above , ranging from level with a bleeding disease.

[Diagnosis]

More maternal, menstruation and more than one of the uterus and increased endometrial thickening normal or individual with a polyp, but the majority of normal pathological examination, a few shows that proliferation can be diagnosed as hypertrophy of the uterus. Should pay attention to and identification of uterine fibroids, especially for a single nuclear intramural muscle or under the mucous membrane, increasing its Palace uniform, and often not easy identification of the uterus mast, scraping through consultation and exploration of intrauterine B-examination can help diagnosis. But there are still a small number of cases in order to laparotomy when diagnosed.

In addition, attention should be paid with adenomyosis, endometrial cancer and other diseases identification.

[Treatment]

Chinese medicine treatment can control menorrhagia, and improve the general condition; androgen therapy can reduce blood volume. Conservative treatment fails, may consider hysterectomy. Ovarian normal under the age of 50, should be retained.

[ET]

(A) more maternal chronic uterine involution incomplete: more women within the myometrium elastic fibers in smooth muscle and blood vessels around the inter-proliferation, causing uterine mast.

(B) of ovarian dysfunction: continuing to stimulate estrogen can myometrium hypertrophy. On the clinical features of the common uterine bleeding patients, especially longer course, have different levels of the uterus increases.

(C) cause inflammation: slow Annex Yan, pelvic connective tissue inflammation and chronic myositis uterus, uterine muscle, caused Collagen fiber proliferation, uterine fibrosis.

(D) pelvic bleeding, uterine caused connective tissue proliferation, can also be caused by uterine mast.

(5) myometrium vascular sclerosis: primary vascular diseases, such as the uterus.

[Pathological changes:

The disease is the basic pathological changes in the uterine muscle and smooth muscle cells in the vessel wall.

(A) general view: a uniform increase of the uterus, muscle hypertrophy of 2.5 ~ 3.2 cm. cut off-white or pink, hardness increase, a woven-fiber arrangement. 1 / 3 muscle vascular eminence, normal or endometrial thickening, and sometimes that the merger of small leiomyoma (diameter of less than 1 cm) or endometrial polyps.

(B) examination: Image see inconsistencies, the following form: ① simply smooth muscle hypertrophy. Microscopy and normal myometrium the same, no collagen fibers hyperplasia, there is no significant change in vessel walls; ② myometrium in the collagen fibers hyperplasia, a uterine fibrosis; ③ muscle within the vessel wall changes: arteriovenous significant expansion , The new blood vessels around the success of the mission fibroelastosis.

[Clinical]

The main symptoms are excessive menstrual volume, continued extension of the number of days; some performance for the period shortened to about 20 days, the number of days of continuous and no significant change or extend the period of performance for the period, but the small amount.

For many patients with the maternal, and the majority of three-over. Sick a long time and who was more bloodshed in anemia Maung gynaecological examinations increased uterine uniform, the general size for the six weeks of pregnancy, a small number of more than eight weeks of pregnancy size, texture more tenacious. Bilateral ovary can be slightly increased, there are multiple follicular cysts.

[Prevention]

As are many reasons for the disease, and some can prevent its occurrence, such as doing a good job of family planning, prevention of post-natal infection, post-partum uterine contraction delinquents should be given timely uterine contraction drugs. Appropriate attention to post-natal prone or supine knee-chest and postnatal movement to prevent the uterus after down, to reduce pelvic congestion. Positive treatment of ovarian dysfunction, to avoid hormones, such as the continuing stimulus.

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